Merit/Imidaclprid is absolutely a systemic insecticide. The inventor (Bayer AG) describes it as such.
http://www.beekeeping.org/articles/us/imidacloprid_bayer.htm
It is a neonicotinoid pesticide.
Pesticides, specifically neonicotinoid pesticides, including imidacloprid, clothianiden and thiamethoxam, poison the bee while it is in the process of collecting nectar and pollen. The poisoning may occur when the material is ingested, or it may be transported to the hive where it poisons other bees in the colony.
It is acutely toxic to earthworms, and highly toxic to honeybees.
http://www.pan-uk.org/pestnews/Actives/imidaclo.htm
Quote:
Beneficial insects
The application of imidacloprid by foliar spraying, is highly toxic to honey bees.
Imidacloprid is acutely toxic to earthworms, for example the LC50 (the lethal concentration required to kill 50% of a test population) of the species Eisenia fetida is between 2 and 4 ppm in the soil. At lower concentrations, the activity of the enzyme cellulase in the soil, that allows the break down of plant litter is reduced by imidacloprid concentrations of 0.2 ppm.
Soil application of granular imidacloprid is the most common. This is less harmful than the foliar spray because it is less likely to come into direct contact with non-target insects. However many natural enemies supplement their diet by feeding on plant material. As imidacloprid is systemic it can be translocated to the surface of the plant, increasing the chances of direct contact with insects on the plants. Laboratory tests have also shown that imidacloprid is acutely toxic to a variety of predatory insects including mirid bugs, ladybirds and lacewings19.
Concentrations:
Quote:
Other studies[citation needed] indicated that concentrations were especially high when the plant is young. These would often be of
* 10 to 20 ppb in upper leaves
* 100 to 200 ppb in other leaves
* less than 1.5 ppb in nectar
* 2 to 3 ppb in pollen
Bayer then agreed that the insecticide may cause disorientation of bees at levels above 20 parts per billion of the active ingredient. Recent studies[citation needed] by researchers at the Institut National de la Recherche Agronomique (INRA) suggest that bee behaviour is affected at levels between 3-16 ppb or possibly even 0.5 ppb.
http://en.wikipedia.org/wiki/Imidacloprid_effects_on_bee_population
Target insects:
Quote:
The biological spectrum of imidacloprid covers a broad range of target pests, such as:
sucking insects: aphids, whiteflies, leaf- and planthoppers, thrips, scales, mealy bugs, bugs, psyllids, phylloxera;
coleoptera (beetles): leaf beetles (e.g. Colorado potato beetle, rice water weevil), wireworms, grubs, flea beetles;
others: lepidopterous leafminers, some dipterous pests, termites, locusts, and fleas.
The trademarks for imidacloprid as a termiticide are Hachikusan® (Japan), Premise® (USA, Australia, Asia, world-wide). Under the trademark Advantage® it has been used commercially as a veterinary medicinal product for flea control on cats and dogs in the USA since 1996 and in Europe since 1997.
http://www.beekeeping.org/articles/us/imidacloprid_bayer.htm
Its use was banned in France due to links to bee population decline and neoniconitiod pesticides.
More on bee effects:
Quote:
Imidacloprid exhibits a high oral toxicity to honeybees. The oral lethal dose 50%
(LD50) is observed between 49 and 102 ng per bee (Nauen et al. 2001), 3.7 and 40.9 ng
per bee (Schmuck et al. 2001), 5 ng per bee (Suchail et al. 2001) or 40 and 60 ng per
bee (Suchail et al. 2001). These values correspond to a lethal food concentration ranging
between 0.1 and 1.6 mg kg–1 (Schmuck et al. 2001). The contact LD50 is about 24 ng/
bee at 24 and 48 h (Suchail et al. 2001). The sub-lethal effect of imidacloprid on bees
has not been investigated until recently. New studies have shown that the crucial
functions of bees such as foraging are affected by sub-lethal doses of imidacloprid in
the range from 1 to 20 μg kg–1 and from 0.1 to 2 ng / bee (Pham-Delegue and Cluzeau
1999; Colin and Bonmatin 2000). A dose of 0.1 ng per bee can also induce a decrease
of habituation (Guez et al. 2001).
http://www.biobees.com/library/pesticide...%20Bonmatin.pdf
Quote:
Conclusion
We developed three extraction schemes followed by LC/MS/MS method to detect
imidacloprid from field samples. These analytical methods are designed to reveal (limit
of detection of 0.1 μg kg–1) and quantify (limit of quantification of 1 μg kg–1) very low
concentrations of imidacloprid in soils, plants and pollens. To date, these methods
are the most sensitive methods available to analyse such materials according to good
laboratory practice and quality criteria from the directive 96/23/EC (Bonmatin 2002).
The long persistence, after one and two years, of imidacloprid in soils has been
demonstrated in this study. Retention of imidacloprid in soils, coupled with the ability of sunflowers to recover the insecticide during the next cultivation, clearly explains
the presence of imidacloprid in untreated plants. This situation is also observed
for maize and several weeds or adventitious plants (plants which grow in fields but
which have not been sown). For untreated wheat, rape and barley, imidacloprid is also
recovered to a lesser extent from contaminated soils (Bonmatin et al. 2000b).
Seed treatment using imidacloprid protects plants against insects and is supposed
to vanish before the arrival of pollinator insects. However, a new phenomenon has
been demonstrated. We have shown that the relative amount of imidacloprid reaches
a minimum, then increases in sunflowers from the time of the capitulum formation. As
a consequence, relatively high levels are observed during flowering in the flowering
heads. At this time, the capitulums of sunflowers contain a mean value of 8 μg kg–1 of
imidacloprid. Another study on maize indicates a similar situation. The ascent of imidacloprid
during flowering appears to be general behaviour, due to both enhanced
metabolism and the strong mobilisation of resources for plants producing large
amounts of grains such as sunflowers and maize.
Our data reveals the presence of imidacloprid in pollens with average values of
3 μg kg–1 (sunflowers and maize). Thus, imidacloprid appears to be bioavailable for
bees in fields, in a range of concentrations corresponding to that of sub-lethal effects
on bees and especially concerning the foraging activity (Colin and Bonmatin 2000;
Colin 2001). This risk situation with respect to sunflowers and maize is worsened when
considering (i) the additional toxic action of several imidacloprid metabolites (Nauen
et al. 1998; Oliveira et al. 2000) as well as (ii) the very low concentrations inducing
chronic mortality of bees which are in the 0.1–1 μg kg–1 range (Suchail et al. 2001;
Belzunces 2001).
The commercialisation and the use of Gaucho® on sunflowers have been suspended
in France since 1999 (J.O.R.F. 1999).